This review addresses the pathophysiology and treatment of hemorrhagic shock - a condition
produced by rapid and significant. Loss of intravascular volume which may, lead sequentially to
hemodynamic instability decreases in oxygen delivery decreased,,, Perfusion tissue, hypoxia cellular, organ damage
, death and. Hemorrhagic shock can be rapidly fatal. The primary goals are. To stop the
.Bleeding and to restore circulating blood volume. Resuscitation may well depend on the estimated
severity of, hemorrhage. It now appears that patients with moderate hypotension from bleeding may
benefit by delaying massive fluid resuscitation. Until they reach a definitive care facility. On the other
hand the use, of intravenous fluids crystalloids or colloids,,And blood products can be life saving in
those patients who are in severe hemorrhagic shock. The optimal method of resuscitation. Has not been
clearly established. A hemoglobin level of 7 - 8 g / dl appears to be an appropriate threshold for
transfusion. In critically ill patients with no evidence of tissue hypoxia. However maintaining a, higher
.Hemoglobin level of 10 g / dl is a reasonable goal in actively bleeding patients the, individuals, elderly or
who are at. Risk for myocardial infarction. Moreover hemoglobin concentration, should not be the only
therapeutic guide in actively. Bleeding patients. Instead therapy should, be aimed at restoring
intravascular volume and adequate hemodynamic parameters.
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